The evolution and pathogenic mechanisms of the rice sheath blight pathogen

نویسندگان

  • Aiping Zheng
  • Runmao Lin
  • Danhua Zhang
  • Peigang Qin
  • Lizhi Xu
  • Peng Ai
  • Lei Ding
  • Yanran Wang
  • Yao Chen
  • Yao Liu
  • Zhigang Sun
  • Haitao Feng
  • Xiaoxing Liang
  • Rongtao Fu
  • Changqing Tang
  • Qiao Li
  • Jing Zhang
  • Zelin Xie
  • Qiming Deng
  • Shuangcheng Li
  • Shiquan Wang
  • Jun Zhu
  • Lingxia Wang
  • Huainian Liu
  • Ping Li
چکیده

Rhizoctonia solani is a major fungal pathogen of rice (Oryza sativa L.) that causes great yield losses in all rice-growing regions of the world. Here we report the draft genome sequence of the rice sheath blight disease pathogen, R. solani AG1 IA, assembled using next-generation Illumina Genome Analyser sequencing technologies. The genome encodes a large and diverse set of secreted proteins, enzymes of primary and secondary metabolism, carbohydrate-active enzymes, and transporters, which probably reflect an exclusive necrotrophic lifestyle. We find few repetitive elements, a closer relationship to Agaricomycotina among Basidiomycetes, and expand protein domains and families. Among the 25 candidate pathogen effectors identified according to their functionality and evolution, we validate 3 that trigger crop defence responses; hence we reveal the exclusive expression patterns of the pathogenic determinants during host infection.

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بررسی اثر قارچ کش و قارچ آنتا گونیست علیه Phizoctonia Solani Kuhn عامل بیماری سوختگی غلاف برنج

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Comprehensive analysis of microRNA-Seq and target mRNAs of rice sheath blight pathogen provides new insights into pathogenic regulatory mechanisms

MicroRNAs (miRNAs) are ∼22 nucleotide non-coding RNAs that regulate gene expression by targeting mRNAs for degradation or inhibiting protein translation. To investigate whether miRNAs regulate the pathogenesis in necrotrophic fungus Rhizoctonia solani AG1 IA, which causes significant yield loss in main economically important crops, and to determine the regulatory mechanism occurring during path...

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عنوان ژورنال:

دوره 4  شماره 

صفحات  -

تاریخ انتشار 2013